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The spinal cord is the main relay of the micturition reflex, with the micturition central pattern generator (CPG) located in the sacral spinal cord. However, the information responsible for the execution of the micturition reflex remains unknown. GABA is an important inhibitory transmitter in the spinal cord, and may provide the inhibitory stimulus for the CPG. The central role of GABA receptors (GABA-Rs) in synaptic inhibition suggests a role for these receptors in the micturition reflex. Therefore, the present study investigated the distribution and characteristics of GABA-Rs in the sacral spinal cord, and the effects of intradural pressure elevation and iontophoretic application of GABA agonists on the voiding reflex. The results show that extracellular GABA-Rs are widely expressed in the sacral spinal cord, including the periaqueductal grey (PAG) region. Intravenous GABA injection reduced micturition latency and reduced voiding efficiency. Intravenous baclofen, an agonist of GABA-B receptors, induced a dose-dependent decrease in the micturition latency. Electrical stimulation in the PAG region decreased the micturition latency, and blocked the effects of intravenous GABA injection. In a separate study, intradural pressure elevation and iontophoretic application of the GABA-A agonist muscimol on the reflex evoked by bladder distention increased micturition latency and induced pelvic floor muscle (PFM) activation. These results indicate the involvement of GABA-B receptors in the micturition reflex. Furthermore, it suggests that GABA-B receptors may be involved in the interneuron of the micturition reflex, and also provide a novel physiological mechanism for the regulation of voiding function.Skunk Apologises For Drug Bust By Video
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